You live in a decadent world seduced by money. As such there are so many deceptive messages you are bombarded with about how to eat right. Ever notice most are from carbohydrate companies who shamelessly hype “Low-Fat” as if that’s all the matters? You’ve probably seen that a lot and over time, without questioning, you believe “low-fat” is better than “high-fat.”
“A fit, healthy body—that is the best fashion statement”
― Jess C. Scott
If carbohydrates are good and fats are bad, why does America continue to gain and gain fat? It’s not just adults, it’s in kids too.
Truth: Carbohydrates drive insulin up. Elevated insulin causes fat to be in storage mode and not burning mode. This makes you a hunger machine, making it hard to really fill up.
“A low carbohydrate diet, on the other hand, allows insulin levels to remain low and fat stores to be burned in the context of reduced hunger and cravings.”
Volek, Jeff; Phinney, Stephen. The Art and Science of Low Carbohydrate Living: An Expert Guide to Making the Life-Saving Benefits of Carbohydrate Restriction Sustainable and Enjoyable .
What Happens on a Low-Carb High-Fat Diet?
Simply put, it puts your body in a state of ketosis where your brain is fed from ketones and the rest of your body with fat. Ketones are a superior brain food and fat is a superior body food.
From Jeff Volek and Stphen Phinney’s book, The Art and Science of Low Carbohydrate Living: An Expert Guide to Making the Life-Saving Benefits of Carbohydrate Restriction Sustainable and Enjoyable, we read:
“The second way to define ‘low carbohydrate’ is physiologic – specifically that level below which there is a fundamental shift in your body’s fuel homeostasis (i.e., energy regulation) away from glucose as a primary fuel. This shift is the adaptation of the body’s hormonal set and inter-organ fuel exchange to allow most of your daily energy needs to be met by fat, either directly as fatty acids or indirectly by ketone bodies made from fat. This process begins for most adults when total carbohydrate is restricted to less than 60 grams per day along with a moderate intake of protein. After a few weeks at this level, the primary serum ‘ketone’ (beta-hydroxybutyrate, or B-OHB), rises above 0.5 millimolar (mM). At this ketone level, which is 10-fold higher than that in someone with a daily intake of 300 grams of carbohydrate, the brain begins to derive a substantial portion of its energy needs from B-OHB, resulting in a commensurate reduced need for glucose. With further restriction of carbohydrate below 50 grams per day, the serum B-OHB rises in response to reduced insulin secretion. However, because dietary protein prompts some insulin release, and serum B-OHB itself stimulates insulin release by the pancreas (albeit subtly), adults eating 20 grams of carbohydrate and 75-150 grams per day of protein rarely run serum B -OHB levels above 3 mM. This is in contrast to the response to total starvation (i.e., no dietary carbs or protein) where the serum BOHB levels run as high as 5 mM. This 10-fold range of serum ketones, from 0.5 to 5 mM, is your body’s normal physiological response to varying degrees of dietary carbohydrate and protein restriction. This response range is called ‘nutritional ketosis’, and is associated with metabolic adaptations allowing your body to maintain a stable state of inter-organ fuel homeostasis. This process is dependent on an adequate, albeit minimal, ability of the pancreas to produce insulin in response to dietary protein and serum ketones, thus maintaining serum B-OHB in the range where it replaces much of your body’s (and your brain’s) need for glucose without distorting whole-body acid-base balance. Nutritional ketosis is by definition a benign metabolic state that gives human metabolism the flexibility to deal with famine or major shifts in available dietary fuels. By contrast, ‘diabetic ketoacidosis’ is an unstable and dangerous condition that occurs when there is inadequate pancreatic insulin response to regulate serum B-OHB. This occurs only in type-1 diabetics or in late stage type-2 diabetes with advanced pancreatic burnout. In this setting of deficient insulin, when exogenous insulin is withheld, serum B-OHB levels reach the 15-25 mM range – 5-to-10-fold higher than the levels characteristic of nutritional ketosis. Unfortunately, among the general public and even many health care professionals as well, these two distinct metabolic states tend to be confused one for another. Understanding how different they are is key to being able to capture the many benefits of nutritional ketosis while avoiding the risks in that very small minority of the population subject to developing diabetic ketoacidosis.”
So what does it basically mean? First, no one ever taught you the the body’s health mechanism is dictated on your brain’s fuel supply.
The Dreaded Insulin Resistance